br Materials and methods br Results and discussion br
Materials and methods
Results and discussion
Conclusion This study revealed that aqueous and methanol extracts from H. crinita leaves modulates the activities of ap5 manufacturer (MOA, AChE, BChE and Na+/K+-ATPase) linked to neurodegenerative diseases, as well as inhibit Fe2+-induced lipid peroxidation in isolated rat brain; these biological activities could be part of the possible mechanisms by which the extracts exert their neuroprotective abilities and could be linked to their phenolic constituents. However, the aqueous extract showed higher neuroprotective properties than the methanol extract. These findings have given a clue on the dietary and medicinal importance of H. crinita leaves as an alternative/complementary therapy for the treatment/management of neurodegenerative diseases. Nevertheless, further in vivo and clinical experiments are recommended.
Conflict of interest